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About Genes & Cancer
Our mission is to combine research from the fields of genomics, bioinformatics, and modem genetics to determine how cancer cells overcome growth regulatory pathways. We aim to cover all aspects of oncogenes, growth suppressor and apoptotic genes, and their roles in tumor development.

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The Roles of USP1 in Ewing Sarcoma

Genes & Cancer

February 14, 2024
PRESS RELEASE: A new research paper was published in Genes & Cancer on February 5, 2024, entitled, “Roles of USP1 in Ewing sarcoma.” continue reading »

Mechanistically Based Blood Proteomic Markers in the TGF-β Pathway Stratify Risk of HCC in Patients With Cirrhosis

News

February 9, 2024
PRESS RELEASE: On February 5, 2024, a new research paper was published in Genes & Cancer, entitled, “Mechanistically based blood proteomic markers in the TGF-β pathway stratify risk of hepatocellular cancer in patients with cirrhosis.” continue reading »

Increased BRD4-L and BRD4-S Promotes Chemotherapy Resistance in Ovarian Carcinoma

Genes & Cancer

September 13, 2023
PRESS RELEASE: On September 12, 2023, a new research paper was published in Genes & Cancer, entitled, “Increased expression of BRD4 isoforms long (BRD4-L) and short (BRD4-S) promotes chemotherapy resistance in high-grade serous ovarian carcinoma.” continue reading »

A Novel Chromosomal Abnormality in AML Patient: Case Report and Literature Review

News

July 7, 2023
PRESS RELEASE: On June 28, 2023, a new research paper was published in Genes & Cancer, entitled, “A novel t (5; 17) (q35; q21) associated with t (8; 21) (q22; q22) in a patient with acute myeloid leukemia: case report and review of literature.” continue reading »

Genes & Cancer | VCP/p97 as a Therapeutic Target in KRAS-Mutant Pancreatic Cancer

News

March 24, 2023
PRESS RELEASE: On March 10, 2023, a new research paper was published in Genes & Cancer, entitled, “VCP/p97, a pleiotropic protein regulator of the DNA damage response and proteostasis, is a potential therapeutic target in KRAS-mutant pancreatic cancer.” continue reading »